6 research outputs found

    Sensorless Rotor Position Estimation For Brushless DC Motors

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    Brushless DC motor speed is controlled by synchronizing the stator coil current with rotor position in order to acquire an accurate alignment of stator rotating field with rotor permanent-magnet field for efficient transfer of energy. In order to accomplish this goal, a motor shaft is instantly tracked by using rotating rotor position sensors such as Hall effect sensors, optical encoders or resolvers etc. Adding sensors to detect rotor position affects the overall reliability and mechanical robustness of the system. Therefore, a whole new trend of replacing position sensors with sensorless rotor position estimation techniques have a promising demand. Among the sensorless approaches, Back-EMF measurement and high frequency signal injection is the most common. Back-EMF is an electromotive force, directly proportional to the speed of rotor revolutions per second, the greater the speed motor acquires the greater the Back-EMF amplitude appears against the motion of rotation. However, the detected Back-EMF is zero at start-up and does not provide motor speed information at this instant. There-fore, Back-EMF based techniques are highly unfavourable for low speed application specially near zero. On the other hand, signal injection techniques are comparatively developed for low or near zero motor speed applications and they also can estimate the on-line motor parameters exploiting the identification theory on phase voltages and currents signals. The signal injection approach requires expensive additional hardware to inject high frequency signal. Since, motors are typically driven with pulse width modulation techniques, high frequency signals are naturally already present which can be used to detect position. This thesis presents rotor position estimation by measuring the voltage and current signals and also proposes an equivalent permanent-magnet synchronous motor model by fitting thedata to a position dependent circuit model

    Rheumatoid arthritis - clinical aspects: 134. Predictors of Joint Damage in South Africans with Rheumatoid Arthritis

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    Background: Rheumatoid arthritis (RA) causes progressive joint damage and functional disability. Studies on factors affecting joint damage as clinical outcome are lacking in Africa. The aim of the present study was to identify predictors of joint damage in adult South Africans with established RA. Methods: A cross-sectional study of 100 black patients with RA of >5 years were assessed for joint damage using a validated clinical method, the RA articular damage (RAAD) score. Potential predictors of joint damage that were documented included socio-demographics, smoking, body mass index (BMI), disease duration, delay in disease modifying antirheumatic drug (DMARD) initiation, global disease activity as measured by the disease activity score (DAS28), erythrocyte sedimentation rate (ESR), C reactive protein (CRP), and autoantibody status. The predictive value of variables was assessed by univariate and stepwise multivariate regression analyses. A p value <0.05 was considered significant. Results: The mean (SD) age was 56 (9.8) years, disease duration 17.5 (8.5) years, educational level 7.5 (3.5) years and DMARD lag was 9 (8.8) years. Female to male ratio was 10:1. The mean (SD) DAS28 was 4.9 (1.5) and total RAAD score was 28.3 (12.8). The mean (SD) BMI was 27.2 kg/m2 (6.2) and 93% of patients were rheumatoid factor (RF) positive. More than 90% of patients received between 2 to 3 DMARDs. Significant univariate predictors of a poor RAAD score were increasing age (p = 0.001), lower education level (p = 0.019), longer disease duration (p < 0.001), longer DMARD lag (p = 0.014), lower BMI (p = 0.025), high RF titre (p < 0.001) and high ESR (p = 0.008). The multivariate regression analysis showed that the only independent significant predictors of a higher mean RAAD score were older age at disease onset (p = 0.04), disease duration (p < 0.001) and RF titre (p < 0.001). There was also a negative association between BMI and the mean total RAAD score (p = 0.049). Conclusions: Patients with longstanding established RA have more severe irreversible joint damage as measured by the clinical RAAD score, contrary to other studies in Africa. This is largely reflected by a delay in the initiation of early effective treatment. Independent of disease duration, older age at disease onset and a higher RF titre are strongly associated with more joint damage. The inverse association between BMI and articular damage in RA has been observed in several studies using radiographic damage scores. The mechanisms underlying this paradoxical association are still widely unknown but adipokines have recently been suggested to play a role. Disclosure statement: C.I. has received a research grant from the Connective Tissue Diseases Research Fund, University of the Witwatersrand. All other authors have declared no conflicts of interes
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